Neuropsychiatric manifestations of depression in multiple sclerosis / Michele L. Pucak en Dialogues in Clinical Neuroscience, Año 2007 Vol. 9 - No. 2 (Junio)

Público ISBD [artículo]  inDialogues in Clinical Neuroscience > Año 2007 Vol. 9 - No. 2 (Junio) . - pp. 125–139 Título : Neuropsychiatric manifestations of depression in multiple sclerosis : neuroinflammatory, neuroendocrine, and neurotrophic mechanisms in the pathogenesis of immune mediated depression Tipo de documento: texto impreso Autores: Michele L. Pucak, Autor ; Katherine A. L. Carroll, Autor ; Douglas A. Kerr, Autor Fecha de publicación: 2022 Artículo en la página: pp. 125–139 Idioma : Inglés (eng) Idioma original : Inglés (eng) Palabras clave: Citocina, Eje hipotálamo-pituitario-suprarrenal, Interferón, Tratamiento, Neuroimagen, Inflamación Resumen: Evidence suggests that depression in multiple sclerosis (MS) is largely biologically mediated by some of the same processes involved in the immunopathogenesis of this neurologic disease. In particular, the increase in proinflammatory cytokines, activation of the hypothalamic-pituitary-adrenal (HPA) axis, and reduction in neurotrophic factors that occur in MS may each account for the increased rate of depression seen in MS. Link: ./index.php?lvl=notice_display&id=28666 [artículo] Neuropsychiatric manifestations of depression in multiple sclerosis : neuroinflammatory, neuroendocrine, and neurotrophic mechanisms in the pathogenesis of immune mediated depression [texto impreso] / Michele L. Pucak, Autor ; Katherine A. L. Carroll, Autor ; Douglas A. Kerr, Autor . - 2022 . - pp. 125–139. Idioma : Inglés (eng) Idioma original : Inglés (eng) in Dialogues in Clinical Neuroscience > Año 2007 Vol. 9 - No. 2 (Junio) . - pp. 125–139 Palabras clave: Citocina, Eje hipotálamo-pituitario-suprarrenal, Interferón, Tratamiento, Neuroimagen, Inflamación Resumen: Evidence suggests that depression in multiple sclerosis (MS) is largely biologically mediated by some of the same processes involved in the immunopathogenesis of this neurologic disease. In particular, the increase in proinflammatory cytokines, activation of the hypothalamic-pituitary-adrenal (HPA) axis, and reduction in neurotrophic factors that occur in MS may each account for the increased rate of depression seen in MS. Link: ./index.php?lvl=notice_display&id=28666

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